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Diagnosis, prevention and treatment of hepatorenal syndrome in cirrhosis.
Postgrad Med J. 2008 Dec;84(998):662-70
Salerno F, Gerbes A, Ginès P, Wong F, Arroyo V.
Francesco
Salerno, Department of Internal Medicine, Policlinico IRCCS San Donato,
University of Milan, Via Morandi, 30, 20097 San Donato (MI), Italy.
This e-mail address is being protected from spambots. You need JavaScript enabled to view it
.
Hepatorenal syndrome (HRS) is a
serious complication of end-stage liver disease, occurring mainly in
patients with advanced cirrhosis and ascites, who have marked
circulatory dysfunction,1 as well as in patients with acute liver
failure.2 In spite of its functional nature, HRS is associated with a
poor prognosis,3 4 and the only effective treatment is liver
transplantation. During the 56th Meeting of the American Association
for the Study of Liver Diseases, the International Ascites Club held a
Focused Study Group (FSG) on HRS for the purpose of reporting the
results of an international workshop and to reach a consensus on a new
definition, criteria for diagnosis and recommendations on HRS
treatment.
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Albumin attenuates DNA damage in primary-cultured neurons.
Neurosci Lett. 2008 Nov 8
Baltanás FC, Weruaga E, Valero J, Recio JS, Alonso JR.
Lab.
of Neural Plasticity and Neurorepair, Institute for Neurosciences of
Castilla y León, Universidad de Salamanca, E-37007 Salamanca, Spain.
Human
serum albumin (HSA) is an effective therapeutic agent that protects
neurons after cerebral ischemia or related injuries by means of its
antioxidant capacity. Our aim was to test whether bovine serum albumin
(BSA) might also provide protection, especially against DNA damage. Rat
cortical neurons were cultured in both the presence and absence of BSA.
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